Support our work

Join our work by donating to our group!
With your gift, you can support cutting-edge medical research and help to increase knowledge about metabolism, the mitochondrial and metabolic mechanisms of age-related degenerative diseases, and their treatments. Our goal is to understand why diseases occur in certain tissues and to use this knowledge to develop targeted treatments.
Doing research that makes a difference
Targeted treatments and "metabolic bypass surgery"

We are especially interested in energy metabolism, cellular nutrient metabolism, and diseases related to problems with these. Energy metabolism takes place in the mitochondria of cells. Mitochondrial diseases include heart, eye, muscle, liver, and brain diseases. Some of these manifest as Parkinson's disease, epilepsy, hearing problems, or diabetes. Mitochondrial disease can manifest with any symptoms at any age. These diseases give us clues to understanding, on a general level, why some tissues are spared from disease while others are affected. By understanding the mechanisms, we can develop tools for detecting and treating diseases at an early stage or even before they occur.

Our group is an international pioneer in metabolic reprogramming. This "metabolic bypass surgery" is an effective treatment when the target of treatment has been precisely identified. 

Our key research questions

Right now, we are investigating the following questions about metabolism and mitochondrial diseases:

1. What mechanisms explain the exceptional diversity of mitochondrial diseases, even though mitochondria are found in all our cells?

2. How can understanding these mechanisms help us to transfer or prevent the onset of the disease or halt its progression?

3. Can we learn about common forms of disease by studying mitochondrial diseases that manifest as, for example, Parkinson's disease, cardiomyopathy, retinal degeneration, or diabetes? 

4. How does environmental stress (viruses, heat, lack of food or overeating, toxins such as alcohol and pollution) affect mitochondria and contribute to the onset of disease?

5. How can modifying energy metabolism reduce age-related degenerative diseases and add healthy years to life?

Impact of our latest findings

Recently, our research has provided additional information on the interaction between viruses and metabolism in the onset of severe epilepsy, vitamin B3 deficiency caused by the disease and its treatment, and metabolic bypass surgery as the basis for individualized treatment. Our findings have led to the development of new treatments and diagnostic tools, and thousands of patients around the world have found an explanation for their previously unknown disease. 

The technology developed by our group, which can be used to measure the body's NAD+ (vitamin B3) metabolites from blood and other tissue samples, led to the creation of a start-up, NADMED Ltd, which serves researchers, clinical laboratories, and the pharmaceutical industry in more than 30 countries. 

 

How to make a donation?

The University of Helsinki Funds manages donated funds and directs them to the recipient specified by the donor. Please be in contact with our Fundraising team in case you are making a donation from outside of Finland or have any questions, we are happy to guide you through the process.

Our bank details are as follows:
Nordea: IBAN = FI15 1660 3001 0767 70, BIC = NDEAFIHH 

Recipient: Helsingin yliopiston rahastot (in Finnish)
Message: A. Wartiovaara group, name of the donor and contact information

For donations over 850 euros or more we kindly ask you to send us a Deed of Donation for tax benefit in Finland. 

Kindly send the deed of donation to:  Donor Relations, P.O. Box 53 (Vuorikatu 3), FI-00014 University of Helsinki or by email to  

If you prefer to e-sign the Deed of Donation, please call or send us a message. We are happy to provide further information about donating and the impact of donations. You can reach us by email and tel. 029 41 21650

University of Helsinki has a money collection permit (RA/2020/737), permitted by the National Police Board on 18 June 2020.